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In addition, the expression of RANTES m RNA and protein was also found to be upregulated by ICAM-1 ligation.

More recently, ICAM-1 has been characterized as a site for the cellular entry of human rhinovirus.

ICAM-1 may also participate in a positive feedback loop and compete with ICAM-2 to maintain a proinflammatory environment conducive to leukocyte endothelial transmigration.

At both the m RNA and protein levels of expression, ICAM-1 ligation was found to upregulate ICAM-1’s own expression in a positive-feedback loop.

This gene encodes a cell surface glycoprotein which is typically expressed on endothelial cells and cells of the immune system.

It binds to integrins of type CD11a / CD18, or CD11b / CD18 and is also exploited by rhinovirus as a receptor for entry into respiratory epithelium.

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